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The American Masculinity Podcast is hosted by Timothy Wienecke — licensed psychotherapist, Air Force veteran, and award-winning men's advocate. Real conversations about masculinity, mental health, trauma, fatherhood, leadership, and growth. Each episode offers expert insight and practical tools to help men show up differently — as partners, fathers, friends, and leaders. No yelling. No clichés. Just grounded, thoughtful masculinity for a changing world.

Episode Summary

Most men don’t walk into a room saying their body is the problem. They talk about their anger. Their sleep. The way they can’t relax even when nothing is wrong. But what if the issue isn’t just in their thoughts? What if their body has been holding onto something for years without them realizing it?

In this episode, host Timothy sits down with physical therapist and movement specialist Zac Cupples. Zac’s work lives at the intersection of breathing, body mechanics, and nervous system regulation. From professional athletes to men dealing with chronic pain and stress, he helps people understand how their bodies have adapted to survive and how those same patterns can start working against them.

This conversation explores how tension, posture, and breath are not random. They are learned responses. Over time, stress, trauma, and high-performance environments can condition the body to stay in a constant state of vigilance. Zac explains how this “lack of space” in the body shows up physically and mentally, and why many men are still reacting to environments they are no longer in.

You’ll hear us break down:

  • Breath and behavior: How the way you breathe directly impacts stress, movement, and emotional control.
  • Lack of space: Why tension in the body reflects limited physical and psychological flexibility.
  • Survival patterns: How strategies that once kept you safe can later create pain and dysfunction.
  • Performance vs. recovery: Why high output without intentional recovery leads to long-term breakdown.
  • Sleep and airway health: How poor breathing habits can disrupt sleep and affect overall health.
  • Jaw, posture, and tension: The hidden connections between stress, clenching, and chronic pain.
  • Individual patterns: Why there is no one-size-fits-all fix for movement or recovery.
  • The “fadeaway” mindset: Learning to adapt your strategies as your body and life change.
  • From finite to infinite games: Shifting from fixing problems to building long-term capacity and health.

We also explore the deeper side of masculinity. The drive to be useful. The cost of ego. The challenge of setting boundaries. And the process of evolving from survival-based habits into intentional, sustainable ways of living.

This episode is not about optimizing every detail or chasing the perfect routine. It is about understanding what your body has been through, recognizing the patterns you’ve built, and creating more options in how you move, breathe, and show up in your life.

Guest Information

  • Zac Cupples is a physical therapist and movement specialist with over 15 years of experience. Works at the intersection of breathing, body position, and how the nervous system functions, helping people see how these things connect and affect the way they move and feel.
  • Works with all kinds of people, from NBA players and pro athletes to those dealing with chronic pain, stress, and physical problems.
  • Founded his own practice focused on performance and recovery, where he coaches people to move better, reduce pain, and build a stronger, healthier body over time.
  • Also creates content and teaches online. Shares simple, science-based tips on breathing, movement, and recovery through YouTube and Instagram, breaking down complex ideas so anyone can understand them.
  • Brings together physical therapy, body mechanics, and nervous system work to help people move with less pain, release built-up tension, and perform better day to day.
  • Key focus areas include breathing patterns, posture, injury recovery, stress-related tension, sleep issues, and long-term health.

Note: Zac Cupples appears in this interview in a personal and professional capacity. The views expressed are his own and do not represent any licensing boards, professional associations, or organizations with whom he may be affiliated.

We fact-checked these claims against peer-reviewed research in respiratory physiology, sleep medicine, cardiovascular science, and autonomic neuroscience. Below are the key confirmations and clarifications:

1. Fuller Breathing After Practice and Its Benefits for Movement, Tension Reduction, and Physiological Function

What was said:

“You’ll be able to get much fuller breath than when you started. And that can improve movement, that can reduce tension, it can do a lot of favorable things.”

Status: True

Detail:

Recent studies on diaphragmatic and slow breathing show clear results. After structured breathwork sessions—like slow diaphragmatic breathing or controlled exercises—people take much fuller breaths than their usual shallow ones. They achieve greater lung volume per breath through better diaphragm movement and use. This expands lung capacity right away. It also activates the rest-and-digest nervous system. The shift lowers muscle tension by calming the fight-or-flight response and reducing overall body arousal. It improves oxygenation and core stability. These changes lead to better movement efficiency during exercise. People notice smoother patterns, less tightness, and gains in exercise capacity and daily function.

Sources:

Balban, M. Y., Neri, E., Kogon, M. M., Weed, L., Nouriani, B., Jo, B., ... & Huberman, A. D. (2023). Brief structured respiration practices enhance mood and reduce physiological arousal. Cell Reports Medicine, 4(1).

https://doi.org/10.1016/j.xcrm.2022.100895

Hamasaki, H. (2020). Effects of diaphragmatic breathing on health: a narrative review. Medicines, 7(10), 65.

https://doi.org/10.3390/medicines7100065

Russo, M. A., Santarelli, D. M., & O’Rourke, D. (2017). The physiological effects of slow breathing in the healthy human. Breathe, 13(4), 298–309.

https://doi.org/10.1183/20734735.009817

Toussaint, L., Nguyen, Q. A., Roettger, C., Dixon, K., Offenbächer, M., Kohls, N., ... & Sirois, F. (2021). Effectiveness of progressive muscle relaxation, deep breathing, and guided imagery in promoting psychological and physiological states of relaxation. Evidence-Based Complementary and Alternative Medicine, 2021(1), 5924040.

https://doi.org/10.1155/2021/5924040

Why it matters for men:

This validates breathwork as a zero-cost, instantly accessible practice for building physical resilience and emotional steadiness—without needing equipment, supplements, or waiting to “feel” motivated.

2. Mouth Breathing, Baroreceptors, and Blood Pressure — Direct Correction

What was said:

“If you are more of a mouth breather, uh, your barrow receptors, which are pressure receptors in the nose aren’t getting stimulated, that has negative effects on, um, blood pressure. So blood pressure can be higher.”

Status: Direct correction required.

Detail:

We got this one wrong. Baroreceptors are pressure sensors located in the carotid sinus, aortic arch, and other cardiovascular structures—not in the nose. They regulate blood pressure through autonomic nervous system responses. The mechanism stated in the episode does not reflect how baroreceptors actually function.

The broader point still stands: chronic mouth breathing is associated with elevated blood pressure, but the pathway runs through airway restriction and sleep-disordered breathing, not nasal pressure receptor stimulation. Habitual mouth breathing is linked to upper airway obstruction, which is itself strongly associated with hypertension.

Sources:

Armstrong, M., & Moore, R. A. (2023). Physiology, baroreceptors. In StatPearls [Internet]. StatPearls Publishing.

https://www.ncbi.nlm.nih.gov/books/NBK538172/

Watso, J. C., Cuba, J. N., Boutwell, S. L., Moss, J. E., Bowerfind, A. K., Fernandez, I. M., Cassette, J. M., May, A. M., & Kirk, K. F. (2023). Acute nasal breathing lowers diastolic blood pressure and increases parasympathetic contributions to heart rate variability in young adults. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 325(6), R797–R808.

https://doi.org/10.1152/ajpregu.00148.2023

Bixler, E. O., Vgontzas, A. N., Lin, H. M., Ten Have, T., Leiby, B. E., Vela-Bueno, A., & Kales, A. (2000). Association of hypertension and sleep-disordered breathing. Archives of Internal Medicine, 160(15), 2289–2295.

https://doi.org/10.1001/archinte.160.15.2289

Dempsey, J. A., Veasey, S. C., Morgan, B. J., & O’Donnell, C. P. (2010). Pathophysiology of sleep apnea. Physiological Reviews, 90(1), 47–112.

https://doi.org/10.1152/physrev.00043.2008

Why it matters for men:

The connection between chronic mouth breathing and elevated blood pressure is real and clinically significant. The mechanism just differs from what was stated. Long-term mouth breathing—often tied to jaw tension, hypervigilance, and disrupted sleep—adds an invisible cardiovascular cost that compounds over years.

3. Fast or Shallow Breathing and Anaerobic Metabolism

What was said:

“If you are breathing fast or shallow or you’re not getting that good gas exchange, that might push you more to anaerobic metabolism.”

Status: Directionally correct, conditionally

Detail:

We were directionally right, but the details matter. Poor gas exchange or significantly impaired breathing can reduce oxygen delivery to tissues, which may shift cellular energy production toward anaerobic glycolysis. However, fast breathing alone does not automatically trigger anaerobic metabolism. The shift only becomes relevant when gas exchange is impaired enough to meaningfully affect oxygen delivery. Stress-breathing is its own problem—it drives the physiological threat response—but it is not the same as pushing tissues into anaerobic metabolism.

Sources:

Brinkman, J. E., Toro, F., & Sharma, S. (2023). Physiology, respiratory drive. In StatPearls [Internet]. StatPearls Publishing.

https://www.ncbi.nlm.nih.gov/books/NBK482414/

Powers, K. A., & Dhamoon, A. S. (2023). Physiology, pulmonary ventilation and perfusion. In StatPearls [Internet]. StatPearls Publishing.

https://www.ncbi.nlm.nih.gov/sites/books/NBK539907/

Radak, Z., Zhao, Z., Koltai, E., Ohno, H., & Atalay, M. (2013). Oxygen consumption and usage during physical exercise: the balance between oxidative stress and ROS-dependent adaptive signaling. Antioxidants & Redox Signaling, 18(10), 1208–1246.

https://doi.org/10.1089/ars.2011.4498

Why it matters for men:

The useful point—that breathing patterns affect performance, recovery, and physiology—holds. Overstating it as an automatic anaerobic shift understates what is actually a more nuanced and serious problem: chronic stress-breathing drives the threat response and compounds physiological wear over time, independent of aerobic or anaerobic metabolism.

4. CPAP Therapy, Mental Clarity, and Mortality Risk

What was said:

“There’s a lot of good evidence with CPAP leading to just a lot of profound changes in mental clarity among other things, and it literally prevents you from dying.”

Status: True.

Detail:

CPAP is the first-line treatment for moderate to severe obstructive sleep apnea. Consistent adherence—generally defined as four or more hours per night—is linked to measurable improvements in attention, memory, processing speed, and daytime alertness, with gains typically apparent within three to six months. Large-scale real-world data associate ongoing CPAP use with significantly reduced all-cause mortality compared to discontinuation. The survival benefit grows with greater nightly usage.

Sources:

Antic, N. A., Catcheside, P., Buchan, C., Hensley, M., Naughton, M. T., Rowland, S., ... & McEvoy, R. D. (2011). The effect of CPAP in normalizing daytime sleepiness, quality of life, and neurocognitive function in patients with moderate to severe OSA. Sleep, 34(1), 111–119.

https://doi.org/10.1093/sleep/34.1.111

Pépin, J. L., Bailly, S., Rinder, P., Adler, D., Benjafield, A. V., Lavergne, F., ... & Hornus, P. (2022). Relationship between CPAP termination and all-cause mortality: a French nationwide database analysis. Chest, 161(6), 1657–1665.

https://doi.org/10.1016/j.chest.2022.02.013

Why it matters for men:

In high-vigilance populations—veterans, first responders, men carrying chronic stress—untreated sleep apnea quietly compounds the cost of disrupted sleep, cognitive impairment, and shortened lifespan. CPAP directly addresses this. For men already managing significant physical and psychological load, restoring sleep quality is not a minor quality-of-life improvement. It is a clinical priority.

5. TMJ Overloading as the Body’s Airway Backup System

What was said:

“If the upper airway is compromised, the jaw overloads as a strategy to keep it open.”

Status: True, with one nuance.

Detail:

When the airway begins to collapse during sleep, the brain signals jaw muscles to activate. Jaw thrust and muscle bracing physically widen the airway at the posterior pharynx, resisting obstruction. Research confirms elevated jaw muscle activity in OSA patients during sleep, with bruxism events frequently occurring immediately after apnea events. This reflex is adaptive in mild to moderate obstruction. In severe cases, it is insufficient and stronger interventions are required.

Sources:

Saito, M., Yamaguchi, T., Mikami, S., Watanabe, K., Gotouda, A., Okada, K., ... & Lavigne, G. (2014). Temporal association between sleep apnea–hypopnea and sleep bruxism events. Journal of Sleep Research, 23(2), 196–203.

https://doi.org/10.1111/jsr.12099

Martynowicz, H., Gac, P., Brzecka, A., Poreba, R., Wojakowska, A., Mazur, G., ... & Wieckiewicz, M. (2019). The relationship between sleep bruxism and obstructive sleep apnea based on polysomnographic findings. Journal of Clinical Medicine, 8(10), 1653.

https://doi.org/10.3390/jcm8101653

 

Why it matters for men:

For men waking with jaw pain, tension headaches, and worn mouthguards, this reframes clenching as a defense mechanism rather than a habit. The mouthguard protects the teeth. It does not address the airway problem driving the clenching. Treating the root cause—through CPAP, oral appliances, or jaw-specific exercises—is the more effective clinical path.

6. TMJ Prevalence in Veterans and High-Stress Populations — Clinical Note

What was said:

“I’d say 70% or more of the vets and responders I work with have some of that.”

Status: Clinical observation, not population data.

Detail:

The 70% figure reflects clinical experience in a specific practice population, not a peer-reviewed prevalence statistic. It is flagged here so listeners can weigh it appropriately.

What the research does support: a systematic review of war veterans found that 56% of those with PTSD experienced temporomandibular disorder symptoms, compared with approximately 20% of veterans without war exposure—more than double the rate. The general population prevalence of TMJ disorders is estimated at roughly 12%. Veterans with PTSD develop TMJ at approximately four to five times the general population rate. The mechanism is well-documented: PTSD drives chronic sympathetic activation, which produces bruxism and jaw-loading as physiological stress responses.

If you or someone you care about is in a high-pressure job and experiencing jaw tension, tension headaches, or teeth grinding, ask a provider about it.

Sources:

Yap, A. U., & Natu, V. P. (2019). Temporomandibular disorders and PTSD in war veterans: a systematic review. Journal of Oral Rehabilitation. [Cited in Prestige Veteran Medical Center, 2025.]

https://prestigeveteranmctx.com/temporomandibular-joint-dysfunction-and-veterans-disability/

National Institute of Dental and Craniofacial Research. (2023). Prevalence of TMJD and its signs and symptoms. U.S. Department of Health and Human Services.

https://www.nidcr.nih.gov/research/data-statistics/facial-pain/prevalence

Curran, S. L., Sherman, J. J., Cunningham, L. L., Okeson, J. P., Reid, K. I., & Carlson, C. R. (1995). Physical and sexual abuse among orofacial pain patients: linkages with pain and psychologic distress. Journal of Orofacial Pain, 9(4), 340–346.

Why it matters for men:

TMJ in high-stress male populations is not incidental. It is a measurable, trackable cost of chronic vigilance. The body uses major muscle groups to stabilize the airway and manage threat arousal. Over years, that costs the jaw, the sleep, and the quality of recovery. It is screened for routinely in clinical practice with this population for exactly that reason.

Full Citations / Further Reading

Antic, N. A., Catcheside, P., Buchan, C., Hensley, M., Naughton, M. T., Rowland, S., ... & McEvoy, R. D. (2011). The effect of CPAP in normalizing daytime sleepiness, quality of life, and neurocognitive function in patients with moderate to severe OSA. Sleep, 34(1), 111-119.

https://doi.org/10.1093/sleep/34.1.111

Armstrong M, Moore RA. Physiology, Baroreceptors. [Updated 2023 Mar 6]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2026 Jan-.

https://www.ncbi.nlm.nih.gov/books/NBK538172/

Balban, M. Y., Neri, E., Kogon, M. M., Weed, L., Nouriani, B., Jo, B., ... & Huberman, A. D. (2023). Brief structured respiration practices enhance mood and reduce physiological arousal. Cell Reports Medicine, 4(1).

https://doi.org/10.1016/j.xcrm.2022.100895

Bixler, E. O., Vgontzas, A. N., Lin, H. M., Ten Have, T., Leiby, B. E., Vela-Bueno, A., & Kales, A. (2000). Association of hypertension and sleep-disordered breathing. Archives of internal medicine, 160(15), 2289-2295.

https://doi.org/10.1001/archinte.160.15.2289

Brinkman, J. E., Toro, F., & Sharma, S. (2023). Physiology, respiratory drive. In StatPearls [Internet]. StatPearls Publishing.

https://www.ncbi.nlm.nih.gov/books/NBK482414/

Clederhouse. (2019, February 18). AASM publishes clinical practice guideline on use of PAP therapy for sleep apnea. American Academy of Sleep Medicine – Association for Sleep Clinicians and Researchers. https://aasm.org/clinical-guideline-pap-therapy/#:~:text=The%20guideline%20includes%20two%20good,up%20should%20occur%20following%20PAP

Dempsey, J. A., Veasey, S. C., Morgan, B. J., & O'Donnell, C. P. (2010). Pathophysiology of sleep apnea. Physiological reviews, 90(1), 47-112.

https://doi.org/10.1152/physrev.00043.2008

Hamasaki, H. (2020). Effects of diaphragmatic breathing on health: a narrative review. Medicines, 7(10), 65.

https://doi.org/10.3390/medicines7100065

Martynowicz, H., Gac, P., Brzecka, A., Poreba, R., Wojakowska, A., Mazur, G., ... & Wieckiewicz, M. (2019). The relationship between sleep bruxism and obstructive sleep apnea based on polysomnographic findings. Journal of clinical medicine, 8(10), 1653.

https://doi.org/10.3390/jcm8101653

Pépin, J. L., Bailly, S., Rinder, P., Adler, D., Benjafield, A. V., Lavergne, F., ... & Hornus, P. (2022). Relationship between CPAP termination and all-cause mortality: a French nationwide database analysis. Chest, 161(6), 1657-1665.

https://doi.org/10.1016/j.chest.2022.02.013

Powers, K. A., & Dhamoon, A. S. (2023). Physiology, pulmonary ventilation and perfusion. In StatPearls [Internet]. StatPearls Publishing.

https://www.ncbi.nlm.nih.gov/sites/books/NBK539907/

Radak, Z., Zhao, Z., Koltai, E., Ohno, H., & Atalay, M. (2013). Oxygen consumption and usage during physical exercise: the balance between oxidative stress and ROS-dependent adaptive signaling. Antioxidants & redox signaling, 18(10), 1208-1246.

https://doi.org/10.1089/ars.2011.4498

Russo, M. A., Santarelli, D. M., & O’Rourke, D. (2017). The physiological effects of slow breathing in the healthy human. Breathe, 13(4), 298-309.

https://doi.org/10.1183/20734735.009817

Saito, M., Yamaguchi, T., Mikami, S., Watanabe, K., Gotouda, A., Okada, K., ... & Lavigne, G. (2014). Temporal association between sleep apnea–hypopnea and sleep bruxism events. Journal of sleep research, 23(2), 196-203.

https://doi.org/10.1111/jsr.12099

Toussaint, L., Nguyen, Q. A., Roettger, C., Dixon, K., Offenbächer, M., Kohls, N., ... & Sirois, F. (2021). Effectiveness of progressive muscle relaxation, deep breathing, and guided imagery in promoting psychological and physiological states of relaxation. Evidence‐Based Complementary and Alternative Medicine, 2021(1), 5924040.

https://doi.org/10.1155/2021/5924040

Watso, J. C., Cuba, J. N., Boutwell, S. L., Moss, J. E., Bowerfind, A. K., Fernandez, I. M., Cassette, J. M., May, A. M., & Kirk, K. F. (2023). Acute nasal breathing lowers diastolic blood pressure and increases parasympathetic contributions to heart rate variability in young adults. American journal of physiology. Regulatory, integrative and comparative physiology, 325(6), R797–R808.

 https://doi.org/10.1152/ajpregu.00148.2023